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EEG Biofeedback: A Generalized Approach to Neuroregulation
By Siegfried Othmer, Susan F. Othmer, and David A. Kaiser
To appear in "APPLIED NEUROPHYSIOLOGY & BRAIN BIOFEEDBACK"
Edited by Rob Kall, Joe Kamiya, and Gary Schwartz
Page 7 of 13
Clinical Evidence: Validating the Model
Clinical
application is both the source and the destination of the theories and
models proposed above. Without the surprises and inventiveness inherent
in daily clinical practice, progress toward a comprehensive model for
EEG biofeedback training would have been much slower, and the scope
much narrower. By its very nature a research orientation must make
certain choices and assumptions, and hold certain procedures invariant
throughout the project. This does not allow for such a variety of
approaches to be tried in such a short time. Yet, due to the volume of
clients we were able to see since 1988, we have achieved significant
depth of experience in a number of areas. It is now our goal to share
this experience widely in order to allow it to be integrated with other
approaches and perspectives, and subjected to more rigorous scientific
evaluation and critique.
The list of conditions for which
clinical efficacy of EEG biofeedback has been observed is given in
Table 2, along with the nature of the qualifying evidence (controlled
studies; published outcome studies; single case studies and conference
presentations). Key references are indicated separately at the end of
the chapter. The number of subjects that fall into each category are
estimated as well. No systematic inquiry was under taken to flesh out
this table, so we don't claim that it is complete. All entries relate
only to data of which we have become aware through various means, and
are therefore a lower limit in each case. In our own work, and that of
our affiliates, we have acquired confirming evidence for all of the
conditions listed, with the exception of Lyme disease.
Table 2. EEG Biofeedback Studies
|
ADHD |
 | Control
| | | Outcome
| | | Case History
 | Kotwal, Burns, & Montgomery (1996)
Tansey & Bruner (1983) |
|
|
|
LEARNING DISABILITIES |
 | Control
| | | Outcome
| | | Case History
 | Kade (1995) Tansey (1993) |
|
|
|
DEVELOPMENTAL DELAY |
 | Control
| | | Outcome
| | | Case History
 | Fleischman (1997) |
|
|
|
AUTISM |
 | Control
| | | Outcome
| | | Case History
 | Sichel, Fehmi, & Goldstein (1995)
Cowan (1994) |
|
|
|
TOURETTE'S SYNDROME |
 | Control
| | | Outcome
| | | Case History
 | Tansey (1986) |
|
|
|
EPILEPSY |
 | Control
| | | Outcome
 | Hansen, Trudeau, & Grace (1996)
Andrews, & Schonfeld (1992)
Tozzo, Elfner, & May (1988)
Tansey (1986)
Cott A, Pavloski RP, Black AH (1979)
Quy & Hutt (1979)
Kuhlman (1978)
Sterman (1977)
Kuhlman (1977)
Wyler, Lockard, Ward, & Finch (1976)
Sterman, MacDonald, & Stone (1974)
Sterman & Friar (1972) |
| | | Case History
|
|
|
MULTIPLE SCLEROSIS |
 | Control
| | | Outcome
| | | Case History
 | Walker (1995) |
|
|
|
CHRONIC PAIN, MIGRAINES |
 | Control
| | | Outcome
 | Othmer & Othmer (1994)
Tansey (1991)
Fehmi (1987) |
| | | Case History
|
|
|
IMMUNE DISORDERS |
 | Control
| | | Outcome
 | Schummer (1995) |
| | | Case History
|
|
|
LYME DISEASE |
 | Control
| | | Outcome
| | | Case History
 | Brown (1995)
Kirk (1994) |
|
|
|
PRE-MENSTRUAL SYNDROME (PMS) |
 | Control
| | | Outcome
 | Othmer & Othmer (1994) |
| | | Case History
|
|
|
POST TRAUMATIC STRESS DISORDER |
 | Control
| | | Outcome
 | Manchester(1995) |
| | | Case History
|
|
|
BIPOLAR DISORDER |
 | Control
| | | Outcome
| | | Case History
 | Othmer & Othmer (1995) |
|
|
Italics - Conference Presentation
This list is staggering in the variety of conditions responding
to the training. A comprehensive treatment of the claims for these
conditions cannot be undertaken here. Instead, a subset of conditions
will be reviewed to indicate the breadth of the remediation
accomplished with respect to types of symptoms, and to demonstrate that
the remediation is non-trivial. That is, it may lie quite out of the
range of what can be expected via spontaneous recovery or even, in some
cases, with the standard interventions. Subsequently, an understanding
of these findings will be sought by looking at underlying physiological
mechanisms.
Before proceeding, it may be useful to make
some more qualitative distinctions among the claims being made with
respect to these varied conditions. Such an attempt is shown in Table
3. Here conditions are ranked according to the consistency with which
remediation can be predicted; the completeness of the remediation; the
duration of the training; and the simplicity or complexity of the
protocols to be brought to bear. For entries in this table, the
judgments are entirely our own, and are based on our own clinical
experience.
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